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By W. Allan Walker

This new version of Pediatric Gastrointestinal sickness is devoted to the upkeep of a complete method of the perform of Pediatric Gastroenterology. thought of to be the definitive reference paintings, this fourth variation has been generally reviewed. for this reason, the scale and content material of assorted sections were converted and new chapters were further. particular sections care for body structure and Pathophysiology, scientific Presentation of disorder, medical Manifestations and administration (discussing the Mouth and Esophagus, the tummy and Duodenum, The gut, and The Pancreas), analysis of Gastrointestinal ailments, and ideas of treatment. every one writer, chosen due to their specific services within the box, has supplied an authoritative and finished account in their subject. This quantity s need-to-know details is supported through a various number of tables, illustrations, and images. the 2 volumes include a CD-ROM with the entire textual content and colour models of the illustrations.

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Extra resources for Pediatric Gastrointestinal Disease (2 Volume Set)

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PROTEOLYTIC ENZYMES ECM-degrading proteinases (endopeptidases) include aspartic, cysteine, and serine proteinases and metalloproteinases, each of them composed of several distinct enzymes synthesized and released by both immune and nonimmune cells (Table 3-10). The components of the ECM, including most collagens, fibronectin, elastin, laminin, entactin, and heparan sulfate proteoglycan, are susceptible to the destructive action of these proteases. This action is counterbalanced by the protective activity of a large number of endogenous inhibitors that include α2macroglobulin, serine protease inhibitors (serpins, kunins, and others), cysteine protease inhibitors (kininogens, stefin, cystatin, and calpastatin), and matrix metalloproteinase (MMP) inhibitors (tissue inhibitors of metalloproteinases (TIMP)-1, -2, and -3).

J-chain expression is more prominent in immunoglobulin A2 than in immunoglobulin A1 colonic immunocytes and is decreased in both subclasses associated with inflammatory bowel disease. Gastroenterology 1988;94:1419–25. 14. Westermann J, Bode U. Distribution of activated T cells migrating through the body: a matter of life and death. Immunol Today 1999;20:302–6. 15. Meuwissen SGM, Feltkamp-Vroom TM, DelaRiviere AB, et al. Analysis of the lympho-plasmacytic infiltrate in Crohn’s disease with special reference to identification of lymphocytesubpopulations.

Intestinal inflammation: a complex interplay of immune-non immune cell interactions. Am J Physiol 1997; 273:G769–75. 5. Brandtzaeg P, Halstensen TS, Kett K, et al. Immunobiology and immunopathology of the human gut mucosa: humoral immunity and intraepithelial lymphocytes. Gastroenterology 1989;97:1562–84. 6. Brandtzaeg P, Farstad IN, Haraldsen G. Regional specialization in the mucosal immune system: primed cells do not always home along the same track. Immunol Today 1999;20:267–77. 7. Peters MG, Secrist H, Anders KR, et al.

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