By Gary C. Kanel
The Atlas of Liver Pathology, through Drs. Gary C. Kanel and Jacob Korula, offers the visible information you want to correctly diagnose all sorts of liver illness. geared up via illness kind, it issues out significant histological positive aspects, updates disorder parameters with new pictures and diagrams, and is helping you realize the medical elements of every illness. It offers fast and handy connection with nearly the entire liver issues regularly visible this day. Nine-hundred-plus top quality, full-color photos catch the gross and histological presentation of liver pathology excellent for comparability to the specimens you stumble upon in perform.
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Extra resources for Atlas of Liver Pathology
Periportal interface inflammatory activity is present, with the hepatocytes exhibiting cytoplasmic eosinophilia as a result of prominence of the mitochondria (oxyphilic change); these types of cells are sometimes seen in chronic viral hepatitis. Figure 2-32 Chronic viral hepatitis. Variability in the same field of cell size, cytoplasmic changes, and the degree of inflammation are often characteristic of chronic viral hepatitis. The lobule to the left of the field shows no inflammation; rather, it exhibits diffuse hydropic change of the hepatocytes secondary to liver cell regeneration.
Isoniazid, ketoconazole; see Table 5-5): Many drugs that cause acute hepatitis may also be associated with fulminant hepatitis (confluent submassive or massive hepatic necrosis). Sometimes the degree of portal inflammation in drug-induced fulminant hepatitis is not as prominent as that seen in acute viral infection. 2. Wilson disease: This autosomal recessive inherited disorder associated with increased hepatic copper deposition may rarely present with jaundice, hemolysis, and renal failure and histologically may resemble the features of fulminant hepatitis secondary to the hepatotropic viruses.
Elimination of the infection occurs by a T-cell-medicated cytokine response, and immunity is established by neutralizing antibodies. iii. Vaccination confers immunity in almost 100% of patients. b. Viral hepatitis type E i. 2-kb RNA virus with the RNA genome expressed in ORF proteins (ORF1, ORF2, and ORF3). There are two genotypes (1 and 2) that occur worldwide, mostly in the developing countries. ii. Recently genotypes 3 and 4 have been noted in swine and humans, and genotype 4 has been shown to be a zoonotic source of subclinical infection in developed countries.